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Bioactive Products
Inhibitors Compound Library
A unique collection of 267 Inhibitor natural compounds
Catalog No: Bb221 Inhibitors Compound Library
Screening Details
Size: 1mg/well * 267 Compounds
2mg/well * 267 Compounds
Cat. No. Information
CFN99959 Huperzine A

Huperzine A is a potent, selective and reversible acetylcholinesterase (AChE) inhibitor and has been widely used in China for the treatment of Alzheimer's disease (AD).Huperzine A induces CYP3A4 expression and activation via PXR dependent pathways, may contribute to drug-drug interactions with ligustrazine and oridonin.
CFN99960 Huperzine B

Huperzine B is a efficient inhibitor of human brain AChE, it can enhance ognitive and protect neuro, may be potentially new drug candidates for Alzheimer's disease therapy.
CFN98037 Coniferaldehyde

Coniferaldehyde exerts anti-inflammatory properties by inducing heme oxygenase-1(HO-1), it inhibits LPS-induced apoptosis through the PKC α/β II/Nrf-2/HO-1 dependent pathway in RAW264.7 macrophage cells. Coniferaldehyde can significantly inhibit the growth of viability of strains of Oenococcus oeni.
CFN98038 Sinapaldehyde

Sinapaldehyde has effects against 65 strains of Candida,Sinapaldehyde inhibits prostaglandin synthetase in a dose-dependent way, it dose-dependently inhibits ethyl phenylpropiolate-induced edema of the rat ear, and can inhibit electrically induced contractions of the guinea pig ileum in a dose-dependent way.The reaction of sinapaldehyde and methylpyranoanthocyanin can obtain a new bluish pigment.
CFN99968 Ginsenoside Rg2

Ginsenoside Rg2 has therapeutic potential for type 2 diabetic patients, it also may represent a potential neurorestorative treatment strategy for vascular dementia or other ischemic insults, has protective effects against H2O2-induced injury and apoptosis in H9c2 cells. Ginsenoside Rg2 suppresses the hepatic glucose production via AMPK-induced phosphorylation of GSK3β and induction of SHP gene expression, regulates the 5-HT3A receptors that are expressed in Xenopus oocytes, inhibits nicotinic acetylcholine receptor-mediated Na+ influx and channel activity.