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Natural Products
Catalog No. Information
CFN97223 2,3-Dehydrokievitone

2,3- Dehydrokievitone exhibits strong cytotoxic activity and induces apoptosis efficiently in cancer cells.
CFN98310 2,3-Dihydroxy-12-oleanen-28-oic acid

2,3-Dihydroxy-olean-12-en-28-oic acids show a low cytotoxicity towards several human tumor cell lines.
CFN98680 2,4,5-Trimethoxybenzaldehyde

2,4,5-Trimethoxybenzaldehyde (2,4,5-TMBA) has anti-adipogenic potential, is a natural cyclooxygenase II (COX-2) inhibitor, suppresses the differentiation of preadipocyts into adipocytes at the concentration of 0.5 mM.
CFN70208 2,4,6-Trihydroxybenzoic acid

2,4,6-Trihydroxybenzoic acid may mediate its effects through a CDK- and SLC5A8-dependent pathway contributing to the prevention of colorectal cancer.
CFN99391 2',4'-Dihydroxy-3',6'-dimethoxychalcone

2',4'-Dihydroxy-3',6'-dimethoxychalcone has cytotoxic, anti-inflammatory activity and anti-cholinesterase activities, it has significant NO inhibitory activity.
CFN97463 2,4-Dihydroxyacetophenone

2,4-Dihydroxyacetophenone is a plant metabolite that can act as a qualitative reagent for ferric iron.
CFN70215 2,4-Dihydroxybenzoic acid

2,4-Dihydroxybenzoic acid is found frequently as a pollutant in natural waters and represents a threat to water quality because it is a precursor to the formation of quinones which are highly toxic. It could as a novel eluent in single column anion chromatography. 2,4-Dihydroxybenzoic acid shows strong inhibitory effects against α-glucosidase enzyme with IC50 values of 549 μg/mL .
CFN92400 2,4'-Dihydroxybenzophenone

Reference standards.
CFN97051 2,4-Dihydroxyphenylacetic acid

2,4-Dihydroxyphenylacetic acid is a functional moiety in the toxin molecule and the biological action of spider toxin is explained by direct interaction with an Fe-S center which is known to play an important role for the glutamate binding.
CFN80131 2',5,6',7-Tetrahydroxyflavone

2',5,6',7-Tetrahydroxyflavone inhibited hepatic testosterone 6beta-hydroxylation (CYP3A4) activity with a IC50 of 7.8 microM.